Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/7814
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dc.contributor.authorYavropoulou, M. P.en
dc.contributor.authorKotsa, K.en
dc.contributor.authorGotzamani Psarrakou, A.en
dc.contributor.authorPapazisi, A.en
dc.contributor.authorTranga, T.en
dc.contributor.authorVentis, S.en
dc.contributor.authorYovos, J. G.en
dc.date.accessioned2015-11-24T16:34:32Z-
dc.date.available2015-11-24T16:34:32Z-
dc.identifier.issn1109-3099-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/7814-
dc.rightsDefault Licence-
dc.subjectAdulten
dc.subjectBiological Markers/blooden
dc.subjectBone Density Conservation Agents/*adverse effectsen
dc.subjectCalcifediol/blooden
dc.subjectCalcium/blooden
dc.subjectFemaleen
dc.subjectFibroblast Growth Factors/blooden
dc.subjectHumansen
dc.subjectHydroxycholecalciferols/*adverse effectsen
dc.subjectHyperparathyroidism, Secondary/blood/chemicaen
dc.titleCinacalcet in hyperparathyroidism secondary to X-linked hypophosphatemic rickets: case report and brief literature reviewen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/20688626-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών και Τεχνολογιών. Τμήμα Βιολογικών Εφαρμογών και Τεχνολογιώνel
heal.publicationDate2010-
heal.abstractX-linked dominant hypophosphatemic rickets (XLH) is the most prevalent genetic form of hypophosphatemic rickets. Standard treatment of XLH patients includes long-term administration of phosphate and calcitriol. Treated patients usually respond well to the conventional therapy and demonstrate amelioration of rachitic symptoms and improved growth. However, long-term administration of phosphate and vitamin D preparations is sometimes complicated with nephrocalcinosis, secondary or tertiary hyperparathyroidism and arterial hypertension. We describe a patient with XLH, caused by a rare missense mutation of the PHEX gene. The patient, while under treatment with alphacalcidol and oral phosphate, developed hypercalciuria, nephrocalcinosis, secondary hyperparathyroidism and arterial hypertension. Cinacalcet was added to the therapeutic regimen and the long-term effects on calciotropic parameters and FGF23 levels are herein reported.en
heal.journalNameHormones (Athens)en
heal.journalTypepeer reviewed-
heal.fullTextAvailabilityTRUE-
Appears in Collections:Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά)

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