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dc.contributor.authorPsarropoulou, C.en
dc.contributor.authorAngelatou, F.en
dc.contributor.authorMatsokis, N.en
dc.contributor.authorVeronikis, D. K.en
dc.contributor.authorKostopoulos, G.en
dc.date.accessioned2015-11-24T16:32:13Z-
dc.date.available2015-11-24T16:32:13Z-
dc.identifier.issn0306-3623-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/7528-
dc.rightsDefault Licence-
dc.titleAbsence of Modification in Gaba and Benzodiazepine Binding and in Choline-Acetyltransferase Activity in Brain-Areas of the Epileptic Mutant Mouse Totteringen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondary<Go to ISI>://A1987K229100003-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών και Τεχνολογιών. Τμήμα Βιολογικών Εφαρμογών και Τεχνολογιώνel
heal.publicationDate1987-
heal.abstract1. In the tottering mutant mouse, which suffers from epilepsy and cerebellar ataxia, we examined whether possible changes in GABA, benzodiazepine receptors and choline acetyltransferase (ChAT) activity are implicated in the pathophysiology of these animals. 2. No alteration in GABAA and GABAB binding could be detected in cerebellar membranes of epileptic mice as compared to normal mice. 3. Benzodiazepine receptor density and affinity showed no statistical difference in cerebellar membranes of epileptic and normal mice. 4. The activity of ChAT determined in the cortices of epileptic and normal mice did not differ significantly between the two groups.en
heal.journalNameGen Pharmacolen
heal.journalTypepeer reviewed-
heal.fullTextAvailabilityTRUE-
Appears in Collections:Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά)

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