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  4. Τμήμα Ιατρικής
  5. Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ
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Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/22408
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dc.contributor.authorKalaitzidis, R.en
dc.contributor.authorTsimihodimos, V.en
dc.contributor.authorBairaktari, E.en
dc.contributor.authorSiamopoulos, K. C.en
dc.contributor.authorElisaf, M. S.en
dc.date.accessioned2015-11-24T19:24:03Z-
dc.date.available2015-11-24T19:24:03Z-
dc.identifier.issn1523-6838-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/22408-
dc.rightsDefault Licence-
dc.subjectAdulten
dc.subjectBlood Glucose/analysisen
dc.subjectBlood Pressureen
dc.subjectBody Mass Indexen
dc.subjectCholesterol, LDL/blooden
dc.subjectCreatinine/blooden
dc.subjectElectrolytes/blooden
dc.subjectFemaleen
dc.subjectGreece/epidemiologyen
dc.subjectHeart Rateen
dc.subjectHumansen
dc.subjectHyperinsulinism/complications/metabolismen
dc.subjectHypophosphatemia/epidemiology/*etiologyen
dc.subjectLipids/blooden
dc.subjectMagnesium/blooden
dc.subjectMaleen
dc.subjectMetabolic Syndrome X/complications/*metabolismen
dc.subjectMiddle Ageden
dc.subjectPhosphates/*metabolismen
dc.subjectPhosphorus, Dietary/pharmacokineticsen
dc.subjectSmoking/blooden
dc.subjectUric Acid/blooden
dc.titleDisturbances of phosphate metabolism: another feature of metabolic syndromeen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/15861350-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2005-
heal.abstractBACKGROUND: Despite important recent advances in the understanding of the consequences of metabolic syndrome, its pathophysiological characteristics remain unclear. It has been proposed that disturbances in phosphate metabolism may contribute to the development of this constellation of cardiovascular risk factors. However, there have been insufficient clinical data supporting this hypothesis to date. The aim of our study is to confirm the presence of hypophosphatemia in patients with metabolic syndrome, as well as investigate mechanisms that may underlie the disturbances in phosphate metabolism in this patient group. METHODS: Two hundred fifty-five individuals were enrolled. The diagnosis of metabolic syndrome was based on Adult Treatment Panel III guidelines. Subjects with fewer than 3 criteria served as controls. RESULTS: Patients with metabolic syndrome showed significantly lower phosphate and magnesium levels compared with controls. Because fractional excretion of phosphate was similar in both groups, we assume that hypophosphatemia in patients with metabolic syndrome can be attributed to decreased dietary intake, as well as internal redistribution of this element. Lower magnesium values in the patient group may result from the same mechanisms as lower phosphate levels. In addition, hyperinsulinemia-induced renal magnesium wasting also may be a contributory factor. CONCLUSION: Patients with metabolic syndrome show significantly lower phosphate and magnesium levels compared with healthy individuals. The clinical significance of these disturbances, as well as their importance as targets for preventive or therapeutic interventions, remains to be established.en
heal.journalNameAm J Kidney Disen
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
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