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  5. Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ
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Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/21691
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dc.contributor.authorBatistatou, A.en
dc.contributor.authorArkoumani, E.en
dc.contributor.authorStefanou, D.en
dc.date.accessioned2015-11-24T19:16:39Z-
dc.date.available2015-11-24T19:16:39Z-
dc.identifier.issn0258-851X-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/21691-
dc.rightsDefault Licence-
dc.subjectAdaptor Proteins, Signal Transducingen
dc.subjectAdulten
dc.subjectAgeden
dc.subjectBreast Neoplasms/*metabolism/pathologyen
dc.subjectCarcinoma in Situ/*metabolism/pathologyen
dc.subjectCarcinoma, Intraductal, Noninfiltrating/*metabolism/pathologyen
dc.subjectCarrier Proteinsen
dc.subjectDNA-Binding Proteins/*metabolismen
dc.subjectFemaleen
dc.subjectHumansen
dc.subjectImmunoenzyme Techniquesen
dc.subjectMiddle Ageden
dc.subjectMutS Homolog 2 Proteinen
dc.subjectNeoplasm Proteins/*metabolismen
dc.subjectNuclear Proteinsen
dc.subjectProto-Oncogene Proteins/*metabolismen
dc.titleExpression of mismatch repair proteins in invasive and in situ carcinoma of the breasten
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/15523909-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2004-
heal.abstractBACKGROUND: Genetic instability is a characteristic feature of familial and sporadic breast carcinomas. It is not clear whether defects in the mismatch repair system accompany this instability. The purpose of this study was to explore the expression of two of the proteins encoded by the DNA mismatch repair genes, namely MLH1 and MSH2, in sporadic in situ and invasive breast carcinomas of various types and grades occurring in Greek patients. MATERIALS AND METHODS: MLH1 and MSH2 expression was monitored immunohistochemically in 60 breast carcinomas (20 in situ and 40 invasive). RESULTS-CONCLUSION: Although we did not detect loss of MLH1 or MSH2 expression, we do believe that our data will contribute to a better understanding of the role of the mismatch repair (MMR) system in breast cancer.en
heal.journalNameIn Vivoen
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
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