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dc.contributor.authorKotsia, A. P.en
dc.contributor.authorDimitriadis, G.en
dc.contributor.authorBaltogiannis, G. G.en
dc.contributor.authorKolettis, T. M.en
dc.date.accessioned2015-11-24T19:13:55Z-
dc.date.available2015-11-24T19:13:55Z-
dc.identifier.issn1687-9635-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/21242-
dc.rightsDefault Licence-
dc.titleTorsade de Pointes and Persistent QTc Prolongation after Intravenous Amiodaroneen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.primary10.1155/2012/673019-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/22474460-
heal.identifier.secondaryhttp://downloads.hindawi.com/journals/crim/2012/673019.pdf-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2012-
heal.abstractWe report a case of torsade de pointes after intravenous amiodarone and concurrent hypokalemia. Despite treatment cessation and correction of electrolyte abnormalities, excessive QTc prolongation was noted, which persisted for 14 days. This prolonged course for QTc normalization may be attributed to the high rate of amiodarone loading and concurrent electrolyte disturbances coupled with possible underlying individual variability in pharmacokinetics.en
heal.journalNameCase Report Meden
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
Appears in Collections:Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ

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