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DC Field | Value | Language |
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dc.contributor.author | Tsiara, S. | en |
dc.contributor.author | Elisaf, M. S. | en |
dc.contributor.author | Mikhailidis, D. P. | en |
dc.date.accessioned | 2015-11-24T19:09:58Z | - |
dc.date.available | 2015-11-24T19:09:58Z | - |
dc.identifier.issn | 0003-3197 | - |
dc.identifier.uri | https://olympias.lib.uoi.gr/jspui/handle/123456789/20772 | - |
dc.rights | Default Licence | - |
dc.subject | Arteriosclerosis/etiology/physiopathology | en |
dc.subject | Blood Coagulation | en |
dc.subject | Humans | en |
dc.subject | Risk Factors | en |
dc.subject | Smoking/*adverse effects/physiopathology | en |
dc.subject | Smoking Cessation | en |
dc.subject | Tobacco Smoke Pollution/adverse effects | en |
dc.subject | Vascular Diseases/*etiology/physiopathology | en |
dc.title | Influence of smoking on predictors of vascular disease | en |
heal.type | journalArticle | - |
heal.type.en | Journal article | en |
heal.type.el | Άρθρο Περιοδικού | el |
heal.identifier.secondary | http://www.ncbi.nlm.nih.gov/pubmed/14565627 | - |
heal.identifier.secondary | http://ang.sagepub.com/content/54/5/507.full.pdf | - |
heal.language | en | - |
heal.access | campus | - |
heal.recordProvider | Πανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικής | el |
heal.publicationDate | 2003 | - |
heal.abstract | Beyond the already well-established strong causative relationship with cancer, smoking increases the risk for vascular disease. Smoking may act directly or adversely influence risk factors contributing to the development of vascular disease. Smoking causes endothelial dysfunction, dyslipidemia (decreased high-density lipoprotein cholesterol levels, hypertriglyceridemia and increased oxidation of low-density lipoprotein cholesterol) and platelet activation leading to a prothrombotic state. Smoking increases emerging risk factors (eg, fibrinogen, homocysteine, and high-sensitivity C-reactive protein) and increases insulin resistance and the risk of developing type 2 diabetes mellitus. The beneficial effects of statins and antioxidants (eg, vitamins C and E, beta-carotene) are counteracted by smoking. Smoking-induced alterations in growth factors, adhesion molecules, and even in genes can accelerate the progression of atherosclerosis. The aim of this review is to consider the adverse consequences of smoking on the factors predisposing to vascular disease and to emphasize the beneficial effects of smoking cessation. | en |
heal.journalName | Angiology | en |
heal.journalType | peer-reviewed | - |
heal.fullTextAvailability | TRUE | - |
Appears in Collections: | Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ |
Files in This Item:
File | Description | Size | Format | |
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Tsiara-2003-Influence of smoking.pdf | 2.41 MB | Adobe PDF | View/Open Request a copy |
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