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Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/20225
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dc.contributor.authorBenetatos, L.en
dc.contributor.authorAlymara, V.en
dc.contributor.authorVassou, A.en
dc.contributor.authorBourantas, K. L.en
dc.date.accessioned2015-11-24T19:05:41Z-
dc.date.available2015-11-24T19:05:41Z-
dc.identifier.issn1751-5521-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/20225-
dc.rightsDefault Licence-
dc.subjectAdulten
dc.subjectFemaleen
dc.subjectHodgkin Disease/complications/diagnosis/therapyen
dc.subjectHumansen
dc.subjectLymphoma, Non-Hodgkin/complications/diagnosis/drug therapyen
dc.subjectMaleen
dc.subjectMiddle Ageden
dc.subjectNeoplasms/*complicationsen
dc.subjectRadiotherapy, Adjuvanten
dc.subjectSeminoma/complications/diagnosis/therapyen
dc.subjectTesticular Neoplasms/complications/diagnosis/therapyen
dc.subjectbeta-Thalassemia/*complications/diagnosis/*physiopathology/therapyen
dc.titleMalignancies in beta-thalassemia patients: a single-center experience and a concise review of the literatureen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.primary10.1111/j.1751-553X.2007.00929.x-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/18333849-
heal.identifier.secondaryhttp://onlinelibrary.wiley.com/store/10.1111/j.1751-553X.2007.00929.x/asset/j.1751-553X.2007.00929.x.pdf?v=1&t=h2bog6dh&s=7eecd77d7c2dadb346c9d1c1f846199171014071-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2008-
heal.abstractThalassemia represents the world's most common monogenic disease, characterized by absence of or decreased globin chain production. The lifespan of thalassemia patients has been extended as a result of current supportive treatment. We report three cases of cancer (non-Hodgkin lymphoma, Hodgkin disease, and seminoma) in thalassemic patients. Factors that may contribute to the pathogenesis of cancer seem to be infections and iron overload through mechanisms of oxidative damage; immunomodulation or coexistence of the two diseases may only be coincidental.en
heal.journalNameInt J Lab Hematolen
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
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