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  4. Τμήμα Ιατρικής
  5. Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ
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Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/19802
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dc.contributor.authorMoutsopoulos, H. M.en
dc.contributor.authorCledes, J.en
dc.contributor.authorSkopouli, F. N.en
dc.contributor.authorElisaf, M. S.en
dc.contributor.authorYouinou, P.en
dc.date.accessioned2015-11-24T19:02:27Z-
dc.date.available2015-11-24T19:02:27Z-
dc.identifier.issn0954-6820-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/19802-
dc.rightsDefault Licence-
dc.subjectAcidosis, Renal Tubular/*complications/physiopathologyen
dc.subjectAdulten
dc.subjectFemaleen
dc.subjectHumansen
dc.subjectHypergammaglobulinemia/complicationsen
dc.subjectKidney Calculi/etiologyen
dc.subjectMiddle Ageden
dc.subjectNephrocalcinosis/complications/*etiologyen
dc.subjectSjogren's Syndrome/*complications/physiopathologyen
dc.titleNephrocalcinosis in Sjogren's syndrome: a late sequela of renal tubular acidosisen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/1865172-
heal.identifier.secondaryhttp://onlinelibrary.wiley.com/store/10.1111/j.1365-2796.1991.tb00429.x/asset/j.1365-2796.1991.tb00429.x.pdf?v=1&t=h0m93sfi&s=aa172b105050eb08a275063abeac3c6a1f369c3d-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate1991-
heal.abstractSjogren's syndrome (SS) is an autoimmune exocrinopathy that develops into systemic autoimmune disease in 25% of patients, leading to general complications, one of which is kidney involvement. It presents mainly as interstitial nephritis, disclosed by hyposthenuria, distal renal tubular acidosis (RTA) and diabetes insipidus. We here describe five cases of SS with type-1 RTA (hyperchloraemic metabolic acidosis with an anion gap and alkaline urine pH) who developed nephrolithiasis, nephrocalcinosis and renal insufficiency. Hypercalciuria due to acidosis was the main nephrocalcinosis-prone factor in four patients; four subjects displayed diminished renal concentrating capacity, and two had hypokalaemia.en
heal.journalNameJ Intern Meden
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
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