Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/19777
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dc.contributor.authorZois, C. D.en
dc.contributor.authorKatsanos, K. H.en
dc.contributor.authorKosmidou, M.en
dc.contributor.authorTsianos, E. V.en
dc.date.accessioned2015-11-24T19:02:13Z-
dc.date.available2015-11-24T19:02:13Z-
dc.identifier.issn1876-4479-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/19777-
dc.rightsDefault Licence-
dc.subjectAnti-Inflammatory Agents/adverse effectsen
dc.subjectCentral Nervous System Diseases/etiologyen
dc.subjectCerebrovascular Disorders/etiologyen
dc.subjectColitis, Ulcerative/drug therapy/physiopathologyen
dc.subjectCranial Nerve Diseases/etiologyen
dc.subjectCrohn Disease/drug therapy/physiopathologyen
dc.subjectGastrointestinal Agents/adverse effectsen
dc.subjectHumansen
dc.subjectInflammatory Bowel Diseases/drug therapy/*physiopathologyen
dc.subjectMuscular Diseases/etiologyen
dc.subjectNervous System Diseases/*etiologyen
dc.subjectPeripheral Nervous System Diseases/etiologyen
dc.subjectThromboembolism/etiologyen
dc.titleNeurologic manifestations in inflammatory bowel diseases: current knowledge and novel insightsen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.primary10.1016/j.crohns.2009.10.005-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/21122494-
heal.identifier.secondaryhttp://ac.els-cdn.com/S1873994609001329/1-s2.0-S1873994609001329-main.pdf?_tid=2c644e23eeb87b79c3a7300ae8b6a19f&acdnat=1333698095_9b7d75ec78c03419447bfa2853c77ad0-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2010-
heal.abstractBACKGROUND: Crohn's disease (CD) and ulcerative colitis (UC), widely known as inflammatory bowel diseases (IBD), are thought to result from an inappropriate activation of the mucosal immune system driven by intestinal bacterial flora. METHODS: Although the extraintestinal manifestations of IBD are well documented, the association of IBD with neurologic and neuromuscular involvement is rare and often controversial, with sporadic and conflicting data on its prevalence and spectrum. In addition, a serious number of the latter manifestations may become life-threatening, playing a very important role in disease morbidity. To define the pattern of neurologic involvement in IBD, the most important manifestations in these patients have been reviewed, exploring also their clinical significance. RESULTS: There is evidence that UC and CD can manifest both in the PNS and CNS. Thrombotic complications are common in IBD patients, but cerebral vascular involvement is rare. CONCLUSIONS: Neurologic manifestations in IBD patients are more common than previously estimated and may follow a different pattern of involvement in CD and UC. Small numbers of patients currently preclude a better characterization of the clinical spectrum and a better understanding of pathogenesis.en
heal.journalNameJ Crohns Colitisen
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
Appears in Collections:Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ

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