Please use this identifier to cite or link to this item:
https://olympias.lib.uoi.gr/jspui/handle/123456789/19286Full metadata record
| DC Field | Value | Language |
|---|---|---|
| dc.contributor.author | Golias, C. | en |
| dc.contributor.author | Batistatou, A. | en |
| dc.contributor.author | Bablekos, G. | en |
| dc.contributor.author | Charalabopoulos, A. | en |
| dc.contributor.author | Peschos, D. | en |
| dc.contributor.author | Mitsopoulos, P. | en |
| dc.contributor.author | Charalabopoulos, K. | en |
| dc.date.accessioned | 2015-11-24T18:58:30Z | - |
| dc.date.available | 2015-11-24T18:58:30Z | - |
| dc.identifier.issn | 1543-5180 | - |
| dc.identifier.uri | https://olympias.lib.uoi.gr/jspui/handle/123456789/19286 | - |
| dc.rights | Default Licence | - |
| dc.subject | Cell Adhesion | en |
| dc.subject | Cell Movement | en |
| dc.subject | Cytokines/immunology/physiology | en |
| dc.subject | Endocarditis, Bacterial/immunology/microbiology/*physiopathology | en |
| dc.subject | Endothelium, Vascular/immunology/physiology | en |
| dc.subject | Humans | en |
| dc.subject | Inflammation/immunology/*physiopathology | en |
| dc.subject | Integrins/immunology/*physiology | en |
| dc.subject | Intercellular Adhesion Molecule-1/immunology/physiology | en |
| dc.subject | Leukocytes/immunology/*physiology | en |
| dc.subject | Membrane Proteins/immunology/physiology | en |
| dc.subject | Selectins/classification/immunology/*physiology | en |
| dc.subject | Staphylococcus/immunology/pathogenicity | en |
| dc.subject | Vascular Cell Adhesion Molecule-1/immunology/physiology | en |
| dc.title | Physiology and pathophysiology of selectins, integrins, and IgSF cell adhesion molecules focusing on inflammation. A paradigm model on infectious endocarditis | en |
| heal.type | journalArticle | - |
| heal.type.en | Journal article | en |
| heal.type.el | Άρθρο Περιοδικού | el |
| heal.identifier.primary | 10.3109/15419061.2011.606381 | - |
| heal.identifier.secondary | http://www.ncbi.nlm.nih.gov/pubmed/21892874 | - |
| heal.identifier.secondary | http://informahealthcare.com/doi/abs/10.3109/15419061.2011.606381 | - |
| heal.language | en | - |
| heal.access | campus | - |
| heal.recordProvider | Πανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικής | el |
| heal.publicationDate | 2011 | - |
| heal.abstract | The development of adhesion bonds, either among cells or among cells and components of the extracellular matrix, is a crucial process. These interactions are mediated by some molecules collectively known as adhesion molecules (CAMs). CAMs are ubiquitously expressed proteins playing a central role in controlling cell migration, proliferation, survival, and apoptosis. Besides their key function in physiological maintenance of tissue integrity, CAMs play an eminent role in various pathological processes such as cardiovascular disorders, atherogenesis, atherosclerotic plaque progression and regulation of the inflammatory response. CAMs such as selectins, integrins, and immunoglobulin superfamily take part in interactions between leukocyte and vascular endothelium (leukocyte rolling, arrest, firm adhesion, migration). Experimental data and pathologic observations support the assumption that pathogenic microorganisms attach to vascular endothelial cells or sites of vascular injury initiating intravascular infections. In this review a paradigm focusing on cell adhesion molecules pathophysiology and infective endocarditis development is given. | en |
| heal.journalName | Cell Commun Adhes | en |
| heal.journalType | peer-reviewed | - |
| heal.fullTextAvailability | TRUE | - |
| Appears in Collections: | Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ | |
Files in This Item:
There are no files associated with this item.
This item is licensed under a Creative Commons License
