Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/19284
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dc.contributor.authorGaulard, P.en
dc.contributor.authorKanavaros, P.en
dc.date.accessioned2015-11-24T18:58:29Z-
dc.date.available2015-11-24T18:58:29Z-
dc.identifier.issn0035-2640-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/19284-
dc.rightsDefault Licence-
dc.subjectApoptosis/geneticsen
dc.subjectCytokines/biosynthesisen
dc.subjectGene Expressionen
dc.subjectGenes, Regulatoren
dc.subjectHerpesvirus 4, Human/isolation & purificationen
dc.subjectHodgkin Disease/metabolism/pathology/*physiopathology/virologyen
dc.subjectHumansen
dc.subjectOncogenesen
dc.subjectReed-Sternberg Cells/pathologyen
dc.titlePhysiopathology of Hodgkin's diseaseen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/9781149-
heal.languagefr-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate1998-
heal.abstractReed-Sternberg cell, the neoplastic component of Hodgkin's disease, still remains "intriguante". Despite contreversies, there is more and more evidence for a lymphoid B cell origin of Hodgkin's disease. The role of Epstein-Barr virus, cytokines and (or) oncogenes expression in the pathogeny can be suggested, although the precise mechanisms leading to transformation and (or) accounting for tumour progression are still elusive. Advances in the biology of Hodgkin's disease have led to the distinction between two entities, "classical" Hodgkin's disease and Poppema's paragranuloma, with different clinical aspects. The definition of diagnostic criteria has been also helpful to separate Hodgkin's disease from other lymphomas mimicking Hodgkin's disease such as anaplastic large cell lymphomas?en
heal.journalNameRev Praten
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
Appears in Collections:Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ

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