Please use this identifier to cite or link to this item:
https://olympias.lib.uoi.gr/jspui/handle/123456789/18668Full metadata record
| DC Field | Value | Language |
|---|---|---|
| dc.contributor.author | Siamopoulos, K. C. | en |
| dc.contributor.author | Elisaf, M. S. | en |
| dc.contributor.author | Drosos, A. A. | en |
| dc.contributor.author | Mavridis, A. A. | en |
| dc.contributor.author | Moutsopoulos, H. M. | en |
| dc.date.accessioned | 2015-11-24T18:54:18Z | - |
| dc.date.available | 2015-11-24T18:54:18Z | - |
| dc.identifier.issn | 0770-3198 | - |
| dc.identifier.uri | https://olympias.lib.uoi.gr/jspui/handle/123456789/18668 | - |
| dc.rights | Default Licence | - |
| dc.subject | Acidosis, Renal Tubular/*etiology | en |
| dc.subject | Adult | en |
| dc.subject | Aged | en |
| dc.subject | Antibodies, Antinuclear/blood | en |
| dc.subject | Female | en |
| dc.subject | Humans | en |
| dc.subject | Middle Aged | en |
| dc.subject | Sjogren's Syndrome/blood/*complications | en |
| dc.title | Renal tubular acidosis in primary Sjogren's syndrome | en |
| heal.type | journalArticle | - |
| heal.type.en | Journal article | en |
| heal.type.el | Άρθρο Περιοδικού | el |
| heal.identifier.secondary | http://www.ncbi.nlm.nih.gov/pubmed/1617898 | - |
| heal.identifier.secondary | http://www.springerlink.com/content/y0653r918414l657/fulltext.pdf | - |
| heal.language | en | - |
| heal.access | campus | - |
| heal.recordProvider | Πανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικής | el |
| heal.publicationDate | 1992 | - |
| heal.abstract | Renal tubular acidosis (RTA) is a frequent extraglandular manifestation of Sjogren's syndrome; however, no distinction on the incidence of this renal tubular defect between primary and secondary Sjogren's syndrome has been reported. This study was undertaken in order to define the frequency of RTA and the possible pathogenetic mechanisms in a group of 21 randomly selected primary Sjogren's syndrome patients. RTA was found in 7 (33%) patients. The incomplete type of the disorder was the most frequent. It seems that the etiology of RTA is multifactorial. Renal excretion of monoclonal proteins and the immunologically-induced interstitial inflammation are the main possible factors of this renal tubular defect. | en |
| heal.journalName | Clin Rheumatol | en |
| heal.journalType | peer-reviewed | - |
| heal.fullTextAvailability | TRUE | - |
| Appears in Collections: | Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ | |
Files in This Item:
| File | Description | Size | Format | |
|---|---|---|---|---|
| Siamopoulos-1992-Renal tubular acidos.pdf | 406.31 kB | Adobe PDF | View/Open Request a copy |
This item is licensed under a Creative Commons License
