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  5. Άρθρα σε επιστημονικά περιοδικά ( Ανοικτά) - ΙΑΤ
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Please use this identifier to cite or link to this item: https://olympias.lib.uoi.gr/jspui/handle/123456789/18640
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dc.contributor.authorKalantaridou, S. N.en
dc.contributor.authorZoumakis, E.en
dc.contributor.authorWeil, S.en
dc.contributor.authorLavasidis, L. G.en
dc.contributor.authorChrousos, G. P.en
dc.contributor.authorMakrigiannakis, A.en
dc.date.accessioned2015-11-24T18:54:02Z-
dc.date.available2015-11-24T18:54:02Z-
dc.identifier.issn1040-8363-
dc.identifier.urihttps://olympias.lib.uoi.gr/jspui/handle/123456789/18640-
dc.rightsDefault Licence-
dc.subjectAntigens, CD95/immunologyen
dc.subjectApoptosis/immunologyen
dc.subjectCorticotropin-Releasing Hormone/*physiologyen
dc.subjectDecidua/cytology/immunologyen
dc.subjectEmbryo Implantation/*immunologyen
dc.subjectFas Ligand Protein/immunologyen
dc.subjectFemaleen
dc.subjectHumansen
dc.subjectHypothalamo-Hypophyseal System/physiologyen
dc.subject*Immune Toleranceen
dc.subjectMaternal-Fetal Exchange/physiologyen
dc.subjectPituitary-Adrenal System/physiologyen
dc.subjectPlacenta/immunologyen
dc.subjectPregnancyen
dc.subjectReceptors, Corticotropin-Releasing Hormone/physiologyen
dc.subjectReproduction/*physiologyen
dc.subjectT-Lymphocytes/immunologyen
dc.titleReproductive corticotropin releasing hormone, implantation, and fetal immunotoleranceen
heal.typejournalArticle-
heal.type.enJournal articleen
heal.type.elΆρθρο Περιοδικούel
heal.identifier.primary10.1080/10408360701548468-
heal.identifier.secondaryhttp://www.ncbi.nlm.nih.gov/pubmed/17943493-
heal.identifier.secondaryhttp://informahealthcare.com/doi/abs/10.1080/10408360701548468-
heal.languageen-
heal.accesscampus-
heal.recordProviderΠανεπιστήμιο Ιωαννίνων. Σχολή Επιστημών Υγείας. Τμήμα Ιατρικήςel
heal.publicationDate2007-
heal.abstractThe fundamental process of implantation involves a series of steps leading to effective cross-talk between invasive trophoblast cells and the maternal endometrium. The molecular interactions at the embryo-maternal interface during the time of blastocyst adhesion and subsequent invasion are not fully understood. Embryonic trophoblast and maternal decidual cells produce corticotropin-releasing hormone (CRH) and express Fas ligand (FasL), a proapoptotic cytokine. Fas and its ligand are pivotal in the regulation of immune tolerance. Trophoblast and decidual CRH play crucial roles in implantation, as well as in the anti-rejection process that protects the fetus from the maternal immune system, primarily by killing activated T cells through Fas-FasL interaction. The potential use of CRH antagonists is presently under intense investigation. CRH antagonists have been used experimentally to elucidate the role of CRH in blastocyst implantation and invasion, early fetal immunotolerance, and premature labor.en
heal.journalNameCrit Rev Clin Lab Scien
heal.journalTypepeer-reviewed-
heal.fullTextAvailabilityTRUE-
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